What is Fever?
Fever, medically termed pyrexia, is defined as an elevation in the body’s core temperature above the normal range. This physiological response is orchestrated by the hypothalamus, the body’s thermostat, which adjusts the ‘set-point’ temperature upwards. This increase in body temperature is typically a defense mechanism triggered by various factors, including infections (bacterial, viral, fungal), inflammatory conditions, malignancies, and autoimmune disorders. These underlying processes prompt the release of immune mediators, signaling the hypothalamus to raise the core temperature as part of the body’s defense strategy.
While a normal body temperature generally fluctuates around 98.6°F (37°C), it can vary slightly throughout the day due to factors like metabolic rate, hormonal changes, and physical activity. However, fever represents a more pronounced and sustained increase beyond these normal daily variations. Fevers are commonly categorized based on their severity:
Category | Temperature |
---|---|
Low-grade fever | 99.1 to 100.4 ºF (37.3 to 38.0 ºC) |
Moderate-grade fever | 100.6 to 102.2 ºF (38.1 to 39.0 ºC) |
High-grade fever | 102.4 to 105.8 ºF (39.1 to 41.0 ºC) |
Hyperthermia/Hyperpyrexia | Greater than 105.8 ºF (Greater than 41.0 ºC) |
It is crucial to distinguish between fever and hyperthermia (sometimes referred to as hyperpyrexia), as they represent different physiological processes and require distinct management approaches. In fever, the hypothalamus actively raises the body’s temperature set-point in response to internal signals, allowing for a controlled temperature elevation. Conversely, hyperthermia occurs when the body temperature rises uncontrollably, exceeding the hypothalamic set-point, often due to external factors such as heat exposure or impaired heat dissipation. Unlike fever, hyperthermia is not regulated by the hypothalamus and poses a greater risk of organ damage.
Category | Fever | Hyperthermia (Hyperpyrexia) |
---|---|---|
Definition | A controlled elevation in body temperature regulated by the hypothalamus in response to infections or inflammation. | An uncontrolled and excessive rise in body temperature that surpasses the hypothalamic set-point and is not regulated by the hypothalamus. |
Mechanism | – The hypothalamus elevates the temperature set-point. – The body generates and conserves heat to reach the new, higher set-point. | – Excessive heat production or impaired heat dissipation mechanisms. – Body temperature increases beyond the hypothalamic regulation. |
Causes | – Infections (bacterial, viral, fungal) – Inflammatory conditions – Certain medications – Immune responses | – Heatstroke – Severe dehydration – Excessive physical exertion in hot environments – Certain drugs or toxins |
Body’s Regulation | – Actively regulated by the hypothalamus. – Employs mechanisms such as sweating and vasodilation to achieve the adjusted temperature set-point. | – Not regulated by the hypothalamus. – Impaired heat dissipation mechanisms, such as reduced sweating or vasodilation. |
Symptoms | – Elevated body temperature (typically 100.4°F to 104°F or 38°C to 40°C) – Chills and shivering – Sweating – Flushed skin – Increased heart and respiratory rate | – Extremely high body temperature (often exceeding 104°F or 40°C) – Hot, dry skin (classic in heatstroke) – Rapid, strong pulse – Confusion, agitation – Seizures or coma in severe cases |
Risks | – Discomfort and potential for febrile seizures, particularly in children – Increased metabolic demands – Risk of dehydration | – Organ dysfunction (e.g., brain, kidneys, liver) – Heatstroke – Potentially fatal if not promptly treated – Neurological damage |
Management | – Identify and treat the underlying cause (e.g., antibiotics for bacterial infections) – Antipyretics (e.g., acetaminophen, ibuprofen) – Encourage hydration – Physical cooling methods if needed | – Immediate and aggressive cooling measures (e.g., ice packs, cooling blankets, evaporative cooling) – Rapid intravenous fluid administration – Monitor and support organ function – Emergency medical intervention is often required |
Comparison of Fever and Hyperthermia
Nursing Diagnosis for Fever
Formulating accurate nursing diagnoses for patients with fever is paramount for effective care planning. These diagnoses are derived from a comprehensive nursing assessment and the nurse’s clinical judgment, tailored to the individual patient’s condition and needs. While the specific application of nursing diagnoses may vary across healthcare settings, the nurse’s expertise is central to developing a patient-centered care plan. Common nursing diagnoses associated with fever include:
- Ineffective Thermoregulation related to an elevated body temperature, as manifested by an increased core temperature, flushed skin, and shivering secondary to an infectious process (specify the infection, if known). This diagnosis highlights the body’s struggle to maintain thermal balance in the presence of fever.
- Fluid Volume Deficit related to excessive fluid loss through diaphoresis (sweating) and increased metabolic demand, as evidenced by dry mucous membranes, decreased urine output, and tachycardia. Fever significantly increases the risk of dehydration, making this diagnosis crucial for preventing complications.
Thermometer for fever assessment
Goals and Expected Outcomes
The primary goals of nursing care for a patient experiencing fever are aimed at alleviating symptoms, addressing the underlying cause, and preventing potential complications. Expected outcomes for patients with fever include:
- Temperature Regulation: The patient’s body temperature will return to within the normal range (96.8-98.6°F or 36-37°C) within a specified timeframe, as appropriate for their clinical condition.
- Symptom Relief: The patient will report a reduction in fever-related discomfort, such as decreased chills, sweating episodes, and muscle or body aches.
- Adequate Hydration: The patient will maintain sufficient fluid intake to prevent dehydration, evidenced by stable vital signs, adequate urine output, and moist mucous membranes.
- Absence of Complications: The patient will exhibit no signs or symptoms of fever-related complications, such as febrile seizures, dehydration, or altered mental status.
Nursing Interventions and Rationales
Nursing interventions for fever management are multifaceted, focusing on reducing body temperature, promoting patient comfort, and preventing complications. These interventions encompass both non-pharmacological and pharmacological approaches, alongside continuous monitoring and environmental adjustments to support thermoregulation.
Nursing Assessment for Fever
1. Accurate Temperature Measurement: Employ a consistent and appropriate method for measuring the patient’s temperature (oral, rectal, tympanic, axillary, temporal artery) based on their age, condition, and clinical setting.
- Rationale: Different measurement sites yield varying temperature readings. Consistency in method and site is crucial for accurate monitoring and trend analysis. Rectal temperatures are generally considered the most accurate, especially in infants and critically ill patients, while oral and axillary methods are less invasive but may be less precise.
2. Establish Baseline Temperature: Inquire about the patient’s usual baseline body temperature or review their medical history for this information.
- Rationale: Individual baseline temperatures can vary. Recognizing a patient’s normal range helps differentiate between typical fluctuations and true fever, preventing misdiagnosis and unnecessary interventions.
3. Validate Subjective Reports: Do not solely rely on patient-reported feelings of warmth or skin palpation to diagnose fever. Always confirm suspected fever with a thermometer reading.
- Rationale: Subjective feelings of warmth and skin palpation are unreliable indicators of fever. Studies have shown palpation to be inaccurate in up to 40% of cases. Objective measurement with a thermometer ensures accurate diagnosis and appropriate management.
4. Select the Optimal Measurement Method: Choose the most appropriate and accurate temperature measurement method based on the patient’s specific condition and clinical needs. For instance, rectal measurement may be preferred for accuracy in infants or critical care settings.
- Rationale: The choice of temperature measurement method impacts accuracy and reliability. Rectal temperatures are considered core temperature measurements and are highly accurate, especially when precision is critical.
5. Document and Monitor Trends: Record the temperature reading, measurement site, and time of assessment meticulously. Monitor temperature trends over time rather than relying on single isolated readings.
- Rationale: Comprehensive documentation allows for the tracking of temperature patterns and responses to interventions. Monitoring trends is more informative than isolated readings, enabling early detection of changes and effective adjustments to the care plan.
6. Assess Skin Condition: Regularly evaluate the patient’s skin for warmth, flushing, diaphoresis (sweating), or night sweats. Conversely, also assess for cold, dry skin, which may paradoxically occur with fever in certain serious conditions.
- Rationale: Skin assessment provides valuable clues about thermoregulation. Warm, flushed skin suggests vasodilation in response to fever, while cold, dry skin may indicate vasoconstriction, potentially signaling a more severe condition like hyperpyrexia or circulatory compromise.
7. Monitor Vital Signs: Frequently monitor temperature, heart rate (pulse), and respiratory rate. Pay close attention to any pulse-temperature dissociation, where the expected heart rate increase with fever is absent or disproportionate, which can be indicative of specific infections like typhoid fever or drug-induced fever.
- Rationale: Vital sign monitoring is crucial in assessing the physiological impact of fever. Typically, heart rate increases by approximately 4.4 beats per minute for every 1°C (1.8°F) increase in core temperature. Deviations from this expected response can provide diagnostic clues.
8. Observe for Heat Conservation Signs: Assess for involuntary muscle contractions (rigors or chills), piloerection (goosebumps), and behaviors like curling up or minimizing body surface area, which are indicative of the body’s attempts to conserve heat and raise core temperature.
- Rationale: These signs reflect the body’s physiological response to raise its temperature set-point during fever. Recognizing these manifestations helps differentiate between the stages of fever and guide appropriate interventions.
9. Fever Pattern Analysis: Monitor and document the pattern of fever (e.g., intermittent, remittent, continuous, relapsing) and note any specific timing, such as diurnal variations (e.g., evening fevers).
- Rationale: Fever patterns can offer diagnostic insights into the underlying cause. For example, persistent evening fevers may be associated with tuberculosis, while cyclic fevers are seen in conditions like malaria.
10. Hyperpyrexia and Neurological Status Monitoring: In patients with severe infections, central nervous system hemorrhage, or heatstroke, be vigilant for hyperpyrexia (extremely high fever, typically >106°F or 41.1°C). Assess for neurological symptoms such as headache, altered mental status, confusion, seizures, or coma.
- Rationale: Hyperpyrexia is a medical emergency that can lead to neurological damage and other organ dysfunction. Prompt identification and intervention are critical to prevent severe complications.
11. Assess for Infection Indicators: Evaluate for signs and symptoms of infection, which is a common cause of fever. These may include elevated white blood cell count, localized signs of infection (redness, swelling, discharge), hypotension, or altered mental status, particularly in hospitalized patients at risk for sepsis.
- Rationale: Fever is often a primary indicator of infection. Identifying the source and nature of infection is essential for targeted treatment. Sepsis, a systemic response to infection, can be life-threatening and requires immediate recognition and management.
12. Hydration Status Assessment: Regularly assess for signs of dehydration, which can be both a cause and a consequence of fever. Monitor fluid intake and output, mucous membrane moisture, skin turgor, and urine concentration. Be mindful of factors that can contribute to fluid loss, such as diaphoresis, tachypnea, and vomiting.
- Rationale: Fever increases metabolic rate and can lead to fluid loss through sweating and increased respiration. Dehydration can exacerbate fever-related discomfort and complications.
13. Inflammatory Response Monitoring: Observe for systemic signs of inflammation, such as elevated erythrocyte sedimentation rate (ESR) or C-reactive protein (CRP), increased white blood cell count, muscle breakdown (elevated creatine kinase), and other acute phase reactants.
- Rationale: Fever is often part of a broader inflammatory response. Monitoring inflammatory markers can help assess the severity and nature of the underlying condition and guide treatment strategies.
General Interventions for Fever Management
1. Promote Oral Fluid Intake: Encourage the patient to increase oral fluid intake, offering water, clear broths, electrolyte-rich beverages (sports drinks, oral rehydration solutions), or diluted fruit juices, as tolerated.
- Rationale: Fever increases fluid losses through perspiration and elevated metabolic rate, increasing the risk of dehydration. Adequate hydration is crucial to maintain circulatory volume, facilitate thermoregulation, and prevent complications.
2. Patient Education on Antipyretics: Educate the patient and/or family members about the purpose, action, proper dosage, frequency, and potential side effects of antipyretic medications (e.g., acetaminophen, ibuprofen). Emphasize the importance of adhering to the prescribed dosing schedule and avoiding exceeding the maximum daily dose.
- Rationale: Informed patients are more likely to adhere to treatment plans and use medications safely and effectively. Education empowers patients to participate actively in their care and recognize when to seek further medical advice.
3. Environmental Assessment for Heat Loss Factors: Evaluate the patient’s immediate environment for factors that may contribute to excessive heat loss, such as drafts, cold surfaces (windows, examination tables), and damp clothing or bed linens.
- Rationale: Environmental factors can significantly impact thermoregulation, especially in vulnerable populations like infants, children, and the elderly. Minimizing heat loss helps prevent shivering and discomfort and supports the body’s efforts to regulate temperature.
4. Room Temperature Adjustment: Ensure the patient’s room is comfortably cool and well-ventilated. Adjust room temperature to a level that promotes heat dissipation without causing shivering. Utilize fans to increase air circulation, if appropriate and tolerated by the patient.
- Rationale: A cooler room environment facilitates heat loss through convection and radiation. Increased air circulation enhances evaporative cooling. However, avoid excessive cooling that could induce shivering, which paradoxically increases heat production.
5. Minimize Conduction Heat Loss: Cover cold surfaces, such as examination tables or weighing scales, with warmed blankets or towels before placing the patient on them.
- Rationale: Direct contact with cold surfaces can cause heat loss through conduction, especially in infants and young children who have a larger surface area to body mass ratio. Insulation with warmed materials minimizes conductive heat loss.
6. Promote Rest and Reduce Activity: Advise the patient to rest and minimize physical activity during fever episodes. Encourage periods of quiet rest and limit strenuous activities.
- Rationale: Physical exertion increases metabolic rate and heat production, which can exacerbate fever and discomfort. Rest reduces metabolic demands, conserves energy, and supports the body’s recovery process.
Pharmacological Interventions for Fever
1. Administer Antipyretic Medications: Administer antipyretic medications, such as acetaminophen (paracetamol) or nonsteroidal anti-inflammatory drugs (NSAIDs) like ibuprofen or naproxen, as prescribed or ordered by the healthcare provider.
- Rationale: Antipyretics reduce fever by inhibiting prostaglandin synthesis in the hypothalamus, thereby lowering the body’s temperature set-point. Acetaminophen and NSAIDs are commonly used and effective antipyretics.
2. Monitor Antipyretic Effectiveness: Regularly reassess the patient’s body temperature following antipyretic administration to evaluate the medication’s effectiveness and the duration of its effect. Document temperature response and any adverse reactions.
- Rationale: Monitoring ensures that the antipyretic medication is achieving the desired temperature reduction and helps guide decisions about repeat dosing or alternative interventions. It also allows for early detection of any adverse drug effects.
Effective nursing care plans are essential for managing fever and related symptoms.
Recommended Resources
For further in-depth information on nursing diagnoses and care planning, consider these valuable resources:
Recommended Nursing Diagnosis and Care Plan Books:
- Ackley and Ladwig’s Nursing Diagnosis Handbook: An Evidence-Based Guide to Planning Care – An evidence-based guide offering a three-step system for client assessment, nursing diagnosis, and care planning.
- Nursing Care Plans – Nursing Diagnosis & Intervention (10th Edition) – Features over 200 care plans reflecting current evidence-based guidelines, including new content on ICNP diagnoses and LGBTQ health issues.
- Nurse’s Pocket Guide: Diagnoses, Prioritized Interventions, and Rationales – A quick-reference tool for identifying correct diagnoses and planning patient care, with over 400 disorders covered.
- Nursing Diagnosis Manual: Planning, Individualizing, and Documenting Client Care – A comprehensive manual for planning, individualizing, and documenting care for over 800 diseases and disorders, including prioritized interventions and rationales.
- All-in-One Nursing Care Planning Resource – E-Book: Medical-Surgical, Pediatric, Maternity, and Psychiatric-Mental Health – Includes over 100 care plans across various specialties, emphasizing interprofessional patient problem focus.
See Also
Explore these additional resources on our site for related nursing care information:
Hyperthermia Nursing Care Plans
References and Sources
- Barnason, S., Williams, J., Proehl, J., Brim, C., Crowley, M., Leviner, S., … & Papa, A. (2012). Emergency nursing resource: non-invasive temperature measurement in the emergency department. Journal of Emergency Nursing, 38(6), 523-530.
- Brody, G. M. (1994). Hyperthermia and hypothermia in the elderly. Clinics in geriatric medicine, 10(1), 213-229.
- Fink, E. L., Kochanek, P. M., Clark, R. S., & Bell, M. J. (2012). Fever control and application of hypothermia using intravenous cold saline. Pediatric critical care medicine: a journal of the Society of Critical Care Medicine and the World Federation of Pediatric Intensive and Critical Care Societies, 13(1), 80.
- Hostler, D., Northington, W. E., & Callaway, C. W. (2009). High-dose diazepam facilitates core cooling during cold saline infusion in healthy volunteers. Applied Physiology, Nutrition, and Metabolism, 34(4), 582–586. doi:10.1139/h09-011
- Isaak, R. S., & Stiegler, M. P. (2016). Review of crisis resource management (CRM) principles in the setting of intraoperative malignant hyperthermia. Journal of anesthesia, 30(2), 298-306.
- Isaak, R. S. (2016). Malignant hyperthermia: case report. Reactions, 1599, 130-30.
- O’Connor, J. P. (2017). Simple and effective method to lower body core temperatures of hyperthermic patients. The American journal of emergency medicine, 35(6), 881-884.
- Reifel Saltzberg, J. M. (2013). Fever and Signs of Shock. Emergency Medicine Clinics of North America, 31(4), 907–926. doi:10.1016/j.emc.2013.07.009
- Schneiderbanger, D., Johannsen, S., Roewer, N., & Schuster, F. (2014). Management of malignant hyperthermia: diagnosis and treatment. Therapeutics and clinical risk management, 10, 355.
- Sessler, D. I., Lee, K. A., & McGuire, J. (1991). Isoflurane anesthesia and circadian temperature cycles in humans. Anesthesiology, 75(6), 985-989.
- Tayefeh, F., Plattner, O., Sessler, D. I., Ikeda, T., & Marder, D. (1998). Circadian changes in the sweating to-vasoconstriction interthreshold range. Pflügers Archiv: European Journal of Physiology, 435(3).